TP53 mutations in myeloid malignancies are either homozygous or hemizygous due to copy number-neutral loss of heterozygosity or deletion of 17p

M. Jasek, L. P. Gondek, N. Bejanyan, R. Tiu, J. Huh, K. S. Theil, C. O'Keefe, M. A. McDevitt, J. P. MacIejewski

Research output: Contribution to journalLetterpeer-review

Original languageEnglish (US)
Pages (from-to)216-219
Number of pages4
JournalLeukemia
Volume24
Issue number1
DOIs
StatePublished - Jan 2010

ASJC Scopus subject areas

  • Hematology
  • Oncology
  • Cancer Research

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