Abstract
Copper is a ubiquitously essential nutrient for cells. In this issue of Cell Metabolism, Kim et al. (2010) demonstrate that tissue copper status can be communicated across organ systems. In a mouse model for cardiac copper deficiency, a serum-carried signal mobilized liver and intestinal copper by inducing ATP7A, a copper-exporting ATPase.
Original language | English (US) |
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Pages (from-to) | 343-344 |
Number of pages | 2 |
Journal | Cell Metabolism |
Volume | 11 |
Issue number | 5 |
DOIs | |
State | Published - May 5 2010 |
ASJC Scopus subject areas
- Physiology
- Molecular Biology
- Cell Biology