Role of hemolysis in neonatal jaundice associated with glucose-6-phosphate dehydrogenase deficiency

Daniel S. Seidman; Michal Shiloh; David K. Stevenson; Hendrik J. Vreman; Ido Paz; Rena Gale

doi:10.1016/S0022-3476(95)70177-X

Role of hemolysis in neonatal jaundice associated with glucose-6-phosphate dehydrogenase deficiency

Daniel S. Seidman, Michal Shiloh, David K. Stevenson, Hendrik J. Vreman, Ido Paz, Rena Gale

School of Medicine

Research output: Contribution to journal › Article › peer-review

27 Scopus citations

Abstract

End-tidal carbon monoxide was measured in 108 newborn infants who had been screened for glucose-6-phosphate dehydrogenase (G6PD) deficiency. The mean ± SD end-tidal carbon monoxide did not differ significantly between the G6PD-deficient and the normal neonates, 2.1 ± 0.6 μl/L and 2.0 ± 0.5 μl/L, respectively, within 12 hours of birth and 1.9 ± 1.4 μl/L and 1.5 ± 0.7 μl/L, respectively, at 48 to 72 hours after birth. On the basis of these measurements, hemolysis is not a sufficient explanation for jaundice in G6PD-deficient newborn infants in the transitional period. (J PEDIATR 1995;127:804-6).

Original language	English (US)
Pages (from-to)	804-806
Number of pages	3
Journal	The Journal of pediatrics
Volume	127
Issue number	5
DOIs	https://doi.org/10.1016/S0022-3476(95)70177-X
State	Published - Nov 1995

ASJC Scopus subject areas

Pediatrics, Perinatology, and Child Health

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10.1016/S0022-3476(95)70177-X

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title = "Role of hemolysis in neonatal jaundice associated with glucose-6-phosphate dehydrogenase deficiency",

abstract = "End-tidal carbon monoxide was measured in 108 newborn infants who had been screened for glucose-6-phosphate dehydrogenase (G6PD) deficiency. The mean ± SD end-tidal carbon monoxide did not differ significantly between the G6PD-deficient and the normal neonates, 2.1 ± 0.6 μl/L and 2.0 ± 0.5 μl/L, respectively, within 12 hours of birth and 1.9 ± 1.4 μl/L and 1.5 ± 0.7 μl/L, respectively, at 48 to 72 hours after birth. On the basis of these measurements, hemolysis is not a sufficient explanation for jaundice in G6PD-deficient newborn infants in the transitional period. (J PEDIATR 1995;127:804-6).",

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T1 - Role of hemolysis in neonatal jaundice associated with glucose-6-phosphate dehydrogenase deficiency

AU - Seidman, Daniel S.

AU - Shiloh, Michal

AU - Stevenson, David K.

AU - Vreman, Hendrik J.

AU - Paz, Ido

AU - Gale, Rena

PY - 1995/11

Y1 - 1995/11

N2 - End-tidal carbon monoxide was measured in 108 newborn infants who had been screened for glucose-6-phosphate dehydrogenase (G6PD) deficiency. The mean ± SD end-tidal carbon monoxide did not differ significantly between the G6PD-deficient and the normal neonates, 2.1 ± 0.6 μl/L and 2.0 ± 0.5 μl/L, respectively, within 12 hours of birth and 1.9 ± 1.4 μl/L and 1.5 ± 0.7 μl/L, respectively, at 48 to 72 hours after birth. On the basis of these measurements, hemolysis is not a sufficient explanation for jaundice in G6PD-deficient newborn infants in the transitional period. (J PEDIATR 1995;127:804-6).

AB - End-tidal carbon monoxide was measured in 108 newborn infants who had been screened for glucose-6-phosphate dehydrogenase (G6PD) deficiency. The mean ± SD end-tidal carbon monoxide did not differ significantly between the G6PD-deficient and the normal neonates, 2.1 ± 0.6 μl/L and 2.0 ± 0.5 μl/L, respectively, within 12 hours of birth and 1.9 ± 1.4 μl/L and 1.5 ± 0.7 μl/L, respectively, at 48 to 72 hours after birth. On the basis of these measurements, hemolysis is not a sufficient explanation for jaundice in G6PD-deficient newborn infants in the transitional period. (J PEDIATR 1995;127:804-6).

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Role of hemolysis in neonatal jaundice associated with glucose-6-phosphate dehydrogenase deficiency

Abstract

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