Role of hemolysis in neonatal jaundice associated with glucose-6-phosphate dehydrogenase deficiency

Daniel S. Seidman, Michal Shiloh, David K. Stevenson, Hendrik J. Vreman, Ido Paz, Rena Gale

Research output: Contribution to journalArticle


End-tidal carbon monoxide was measured in 108 newborn infants who had been screened for glucose-6-phosphate dehydrogenase (G6PD) deficiency. The mean ± SD end-tidal carbon monoxide did not differ significantly between the G6PD-deficient and the normal neonates, 2.1 ± 0.6 μl/L and 2.0 ± 0.5 μl/L, respectively, within 12 hours of birth and 1.9 ± 1.4 μl/L and 1.5 ± 0.7 μl/L, respectively, at 48 to 72 hours after birth. On the basis of these measurements, hemolysis is not a sufficient explanation for jaundice in G6PD-deficient newborn infants in the transitional period. (J PEDIATR 1995;127:804-6).

Original languageEnglish (US)
Pages (from-to)804-806
Number of pages3
JournalThe Journal of pediatrics
Issue number5
StatePublished - Nov 1995


ASJC Scopus subject areas

  • Pediatrics, Perinatology, and Child Health

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