TY - JOUR
T1 - Relation of hypertension to changes in the arteries
AU - Heptinstall, Robert H.
N1 - Funding Information:
From the Department of Pathology, the Johns Hopkins University School of Medicine, and the Johns Hopkins Hospital, Baltimore, Md. Supported by USPHS Research Grant HL 07835. Reprint requests should be addressed to Robert H. Heptinstall, M.D., Department of Pathology, the Johns Hopkins Hospital, Baltimore, Md. 21205. 0 1974 by Grune & Stratton, Inc.
PY - 1974
Y1 - 1974
N2 - Hypertension can damage small blood vessels, and this effect is best seen in the arterioles of the kidney, which undergo fibrinoid necrosis. It also has a potentiating effect on arteriosclerosis of the larger vessels such as the coronary, carotid, and vertebral arteries. The relation of hypertension to endarteritis fibrosa of the interlobular arteries of the kidney is not clear; while at one time it was felt that hypertension caused this lesion, recently acquired information raises the possibility that the arterial changes result from intravascular coagulation and may cause or accelerate the hypertension.
AB - Hypertension can damage small blood vessels, and this effect is best seen in the arterioles of the kidney, which undergo fibrinoid necrosis. It also has a potentiating effect on arteriosclerosis of the larger vessels such as the coronary, carotid, and vertebral arteries. The relation of hypertension to endarteritis fibrosa of the interlobular arteries of the kidney is not clear; while at one time it was felt that hypertension caused this lesion, recently acquired information raises the possibility that the arterial changes result from intravascular coagulation and may cause or accelerate the hypertension.
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U2 - 10.1016/0033-0620(74)90035-8
DO - 10.1016/0033-0620(74)90035-8
M3 - Article
C2 - 4365951
AN - SCOPUS:0016195492
SN - 0033-0620
VL - 17
SP - 25
EP - 30
JO - Progress in Cardiovascular Diseases
JF - Progress in Cardiovascular Diseases
IS - 1
ER -