The authors present the case of an infant harboring a vein of Galen arteriovenous malformation with conspicuous cerebral calcifications that progressively regressed after staged endovascular obliteration of the lesion. The role of venous hypertension and hydrocephalus secondary to the arteriovenous shunt are discussed to explain the formation and regression of the cerebral calcifications.
- Endovascular treatment
- Regression of cerebral calcifications
- Vein of Galen arteriovenous malformation
- Venous hypertension
ASJC Scopus subject areas
- Pediatrics, Perinatology, and Child Health
- Clinical Neurology