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Mutated CALR: Tails from the crypt
Jerry L. Spivak
School of Medicine
Research output
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Contribution to journal
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Review article
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peer-review
2
Scopus citations
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Medicine & Life Sciences
Calreticulin
100%
Thrombopoietin Receptors
54%
Gain of Function Mutation
47%
Exome
43%
Endoplasmic Reticulum
34%
Genes
14%
Neoplasms
11%
Chemical Compounds
Mutation
60%
Surface
22%