Monoamine oxidase, phenylethylamine, norepinephrine and schizophrenia

R. J. Wyatt, F. Karoum, D. M. Stoff, J. E. Kleinman, J. C. Gillin, D. V. Jeste, S. G. Potkin

Research output: Contribution to journalArticle

Abstract

The chain of events from decreased MAO activity in the peripheral blood of certain schizophrenic patients, particularly paranoid patients, to increased concentrations of PEA, to increased NE and NE turnover in the brain of schizophrenic patients, may prove not to be as closely linked as the current hypothesis and available data suggest. Nevertheless, the notion that this chain exists is testable and provides an integrated hypothesis of etiology for a subgroup of schizophrenic patients.

Original languageEnglish (US)
Pages (from-to)437-442
Number of pages6
JournalUnknown Journal
Volume19
Issue number5
DOIs
StatePublished - Jan 1 1981

ASJC Scopus subject areas

  • Genetics
  • Genetics(clinical)

Fingerprint Dive into the research topics of 'Monoamine oxidase, phenylethylamine, norepinephrine and schizophrenia'. Together they form a unique fingerprint.

  • Cite this

    Wyatt, R. J., Karoum, F., Stoff, D. M., Kleinman, J. E., Gillin, J. C., Jeste, D. V., & Potkin, S. G. (1981). Monoamine oxidase, phenylethylamine, norepinephrine and schizophrenia. Unknown Journal, 19(5), 437-442. https://doi.org/10.1111/j.1399-0004.1981.tb00741.x