Long-term follow-up of ETV6-RUNX1 ALL reveals that NCI risk, rather than secondary genetic abnormalities, is the key risk factor

A. Enshaei, C. J. Schwab, Z. J. Konn, C. D. Mitchell, S. E. Kinsey, R. Wade, A. Vora, C. J. Harrison, Anthony Moorman

Research output: Contribution to journalLetter

Original languageEnglish (US)
Pages (from-to)2256-2259
Number of pages4
JournalLeukemia
Volume27
Issue number11
DOIs
StatePublished - Nov 1 2013
Externally publishedYes

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ASJC Scopus subject areas

  • Hematology
  • Cancer Research
  • Anesthesiology and Pain Medicine

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Long-term follow-up of ETV6-RUNX1 ALL reveals that NCI risk, rather than secondary genetic abnormalities, is the key risk factor. / Enshaei, A.; Schwab, C. J.; Konn, Z. J.; Mitchell, C. D.; Kinsey, S. E.; Wade, R.; Vora, A.; Harrison, C. J.; Moorman, Anthony.

In: Leukemia, Vol. 27, No. 11, 01.11.2013, p. 2256-2259.

Research output: Contribution to journalLetter

Enshaei, A, Schwab, CJ, Konn, ZJ, Mitchell, CD, Kinsey, SE, Wade, R, Vora, A, Harrison, CJ & Moorman, A 2013, 'Long-term follow-up of ETV6-RUNX1 ALL reveals that NCI risk, rather than secondary genetic abnormalities, is the key risk factor', Leukemia, vol. 27, no. 11, pp. 2256-2259. https://doi.org/10.1038/leu.2013.136
Enshaei, A. ; Schwab, C. J. ; Konn, Z. J. ; Mitchell, C. D. ; Kinsey, S. E. ; Wade, R. ; Vora, A. ; Harrison, C. J. ; Moorman, Anthony. / Long-term follow-up of ETV6-RUNX1 ALL reveals that NCI risk, rather than secondary genetic abnormalities, is the key risk factor. In: Leukemia. 2013 ; Vol. 27, No. 11. pp. 2256-2259.
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