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Dive into the research topics of 'Hemolytically inactive C4B complement allotype caused by a proline to leucine mutation in the C5-binding site'. Together they form a unique fingerprint.- Sort by
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Robert H. McLean, Gary Niblack, Bruce Julian, Tao Wang, Robert Wyatt, John A. Phillips, Timothy S. Collins, Jerry Winkelstein, David Valle
Research output: Contribution to journal › Article › peer-review