Two patients had episcleritis and a secondary open angle glaucoma. Anterior uveitis was absent in three of the four eyes. The low facility of outflow suggests that increased episcleral venous pressure was not the mechanism of the glaucoma. The response of the intraocular pressure and, in one case, the outflow facility to steroid therapy suggests that inflammation of angle structures was the pathogenesis of the raised intraocular pressure.
|Original language||English (US)|
|Number of pages||3|
|Journal||Archives of ophthalmology|
|State||Published - Oct 1975|
ASJC Scopus subject areas