TY - JOUR
T1 - Giant cell arteritis in the ocular ischemic syndrome
AU - Hamed, L. M.
AU - Guy, J. R.
AU - Moster, M. L.
AU - Bosley, T.
N1 - Funding Information:
From the Departments of Ophthalmology (Drs. Hamed andGuy) andNeurology (Dr. Guy), University of Florida College of Medicine, Gainesville, Florida; Departments of Neurology (Dr. Moster) and Ophthalmology (Dr. Moster), Temple University Hospital, Philadelphia, Pennsylvania; and Wills Eye Hospital (Dr. Bosley), Philadelphia, Pennsylvania. This study was supported inpart by anunrestricted departmental grant from Research to Prevent Blindness, Inc.,NewYork, New York.
PY - 1992
Y1 - 1992
N2 - Oculomotor nerve paresis, ocular hypotony, anterior segment ischemia, and the ocular ischemic syndrome are uncommon manifestations of giant cell arteritis. Four patients with these findings had giant cell arteritis documented by temporal artery biopsy. Cerebral angiography or ultrasonography, or both, performed in three patients, excluded hemodynamically significant stenosis of the internal carotid artery as the cause of ocular ischemia and cerebral aneurysms as the cause of oculomotor nerve paresis. Corticosteroid treatment, administered to three patients, resulted in resolution of the oculomotor deficits and the clinical signs of ocular ischemia, although the visual acuity in one patient improved from 20/400 to 20/60. Giant cell arteritis should be considered in the differential diagnosis of the ocular ischemic syndrome.
AB - Oculomotor nerve paresis, ocular hypotony, anterior segment ischemia, and the ocular ischemic syndrome are uncommon manifestations of giant cell arteritis. Four patients with these findings had giant cell arteritis documented by temporal artery biopsy. Cerebral angiography or ultrasonography, or both, performed in three patients, excluded hemodynamically significant stenosis of the internal carotid artery as the cause of ocular ischemia and cerebral aneurysms as the cause of oculomotor nerve paresis. Corticosteroid treatment, administered to three patients, resulted in resolution of the oculomotor deficits and the clinical signs of ocular ischemia, although the visual acuity in one patient improved from 20/400 to 20/60. Giant cell arteritis should be considered in the differential diagnosis of the ocular ischemic syndrome.
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U2 - 10.1016/S0002-9394(14)74798-1
DO - 10.1016/S0002-9394(14)74798-1
M3 - Article
C2 - 1598963
AN - SCOPUS:0026777148
SN - 0002-9394
VL - 113
SP - 702
EP - 705
JO - American journal of ophthalmology
JF - American journal of ophthalmology
IS - 6
ER -