| Original language | English (US) |
|---|---|
| Pages (from-to) | 72-75 |
| Number of pages | 4 |
| Journal | Vascular Medicine (United Kingdom) |
| Volume | 24 |
| Issue number | 1 |
| DOIs | |
| State | Published - Feb 1 2019 |
Keywords
- ERK
- GNAQ
- Gαq
- Sturge–Weber syndrome
- blood vessel
- endothelial cells
- leptomeninges
ASJC Scopus subject areas
- Cardiology and Cardiovascular Medicine
Access to Document
Other files and links
Cite this
- APA
- Standard
- Harvard
- Vancouver
- Author
- BIBTEX
- RIS
Gαq and hyper-phosphorylated ERK expression in Sturge–Weber syndrome leptomeningeal blood vessel endothelial cells. / the BVMC Sturge–Weber syndrome Project Workgroup.
In: Vascular Medicine (United Kingdom), Vol. 24, No. 1, 01.02.2019, p. 72-75.Research output: Contribution to journal › Article › peer-review
}
TY - JOUR
T1 - Gαq and hyper-phosphorylated ERK expression in Sturge–Weber syndrome leptomeningeal blood vessel endothelial cells
AU - the BVMC Sturge–Weber syndrome Project Workgroup
AU - Wellman, Rebecca J.
AU - Cho, Su Bin
AU - Singh, Pratibha
AU - Tune, Miriya
AU - Pardo, Carlos A.
AU - Comi, Anne M.
N1 - Funding Information: The authors disclosed receipt of the following financial support for the research, authorship, and/or publication of this article: this work was supported by grants from the National Institute of Neurological Disorders and Stroke (NINDS) (National Institutes of Health (NIH) U54NS065705) (to Michael Lawton; Sturge– Weber Project to AMC). The Brain Vascular Malformation Consortium (U54NS065705) is a part of the NIH Rare Diseases Clinical Research Network (RDCRN), supported through the collaboration between the NIH Office of Rare Diseases Research (ORDR) at the National Center for Advancing Translational Science (NCATS) and the NINDS. Brain tissue samples were obtained from the NIH Neuro BioBank, the Johns Hopkins Brain Tissue Bank, and the UCSF Brain Tissue Bank. Funding Information: The BVMC Sturge–Weber syndrome Project Workgroup are: Karen L Ball, Sturge–Weber Foundation, Houston, TX, USA; Brian J Fisher, Sturge–Weber Foundation, Houston, TX, USA; Jim Koneig, Division of Neuroscience, National Institute of Neurological Disorders and Stroke, National Institutes of Health, Bethesda, MD, USA; Douglas A Marchuk, Department of Molecular Genetics and Microbiology, Duke University School of Medicine, Durham, NC, USA; Marsha A Moses, Vascular Biology Program, Boston Children’s Hospital, Boston, MA, USA, and Department of Surgery, Harvard Medical School, Boston Children’s Hospital, Boston, MA, USA; Jonathan Pevsner, Department of Neurology, Hugo W Moser Research Institute, Kennedy Krieger Institute, Baltimore, MD, USA, and Department of Psychiatry and Behavioral Sciences, Johns Hopkins School of Medicine, Baltimore, MD, USA. The authors disclosed receipt of the following financial support for the research, authorship, and/or publication of this article: this work was supported by grants from the National Institute of Neurological Disorders and Stroke (NINDS) (National Institutes of Health (NIH) U54NS065705) (to Michael Lawton; Sturge–Weber Project to AMC). The Brain Vascular Malformation Consortium (U54NS065705) is a part of the NIH Rare Diseases Clinical Research Network (RDCRN), supported through the collaboration between the NIH Office of Rare Diseases Research (ORDR) at the National Center for Advancing Translational Science (NCATS) and the NINDS. Brain tissue samples were obtained from the NIH Neuro BioBank, the Johns Hopkins Brain Tissue Bank, and the UCSF Brain Tissue Bank.
PY - 2019/2/1
Y1 - 2019/2/1
KW - ERK
KW - GNAQ
KW - Gαq
KW - Sturge–Weber syndrome
KW - blood vessel
KW - endothelial cells
KW - leptomeninges
UR - http://www.scopus.com/inward/record.url?scp=85053343703&partnerID=8YFLogxK
UR - http://www.scopus.com/inward/citedby.url?scp=85053343703&partnerID=8YFLogxK
U2 - 10.1177/1358863X18786068
DO - 10.1177/1358863X18786068
M3 - Article
C2 - 30112971
AN - SCOPUS:85053343703
VL - 24
SP - 72
EP - 75
JO - Vascular Medicine
JF - Vascular Medicine
SN - 1358-863X
IS - 1
ER -