Abstract
In this issue of Cancer Cell, Zhang et al. reports that the iron-dependent 2-oxoglutarate dioxygenase or prolyl hydroxylase EglN2 induces Cyclin D1 levels, egging on breast tumorigenesis. Their observations through loss of function studies suggest the potential for drug-like molecules inhibiting EglN to serve as new cancer therapeutics.
Original language | English (US) |
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Pages (from-to) | 361-362 |
Number of pages | 2 |
Journal | Cancer cell |
Volume | 16 |
Issue number | 5 |
DOIs | |
State | Published - Nov 6 2009 |
ASJC Scopus subject areas
- Oncology
- Cancer Research
- Cell Biology