Disseminated superficial actinic porokeratosis

Panta Rouhani, Max Fischer, Shane Meehan, Miriam Keltz Pomeranz

Research output: Contribution to journalArticle

Abstract

Disseminated superficial actinic porokeratosis, which was described in 1966, is characterized by small, atrophic patches with distinctive keratin rims that occur on sun-exposed areas of the extremities, shoulders, and back. The diagnosis is based on the histopathologic finding of a cornoid lamella, absence of a granular layer, and often a thin epidermis. It is associated with exposure to ultraviolet radiation. Gene studies suggest a pathway defect in which several mutations in keratinocyte proliferation and differentiation lead to development of porokeratosis.

Original languageEnglish (US)
Pages (from-to)24
Number of pages1
JournalDermatology Online Journal
Volume18
Issue number12
StatePublished - Dec 2012
Externally publishedYes

Fingerprint

Porokeratosis
Solar System
Keratins
Keratinocytes
Epidermis
Extremities
Radiation
Mutation
Genes

ASJC Scopus subject areas

  • Dermatology

Cite this

Rouhani, P., Fischer, M., Meehan, S., & Pomeranz, M. K. (2012). Disseminated superficial actinic porokeratosis. Dermatology Online Journal, 18(12), 24.

Disseminated superficial actinic porokeratosis. / Rouhani, Panta; Fischer, Max; Meehan, Shane; Pomeranz, Miriam Keltz.

In: Dermatology Online Journal, Vol. 18, No. 12, 12.2012, p. 24.

Research output: Contribution to journalArticle

Rouhani, P, Fischer, M, Meehan, S & Pomeranz, MK 2012, 'Disseminated superficial actinic porokeratosis', Dermatology Online Journal, vol. 18, no. 12, pp. 24.
Rouhani P, Fischer M, Meehan S, Pomeranz MK. Disseminated superficial actinic porokeratosis. Dermatology Online Journal. 2012 Dec;18(12):24.
Rouhani, Panta ; Fischer, Max ; Meehan, Shane ; Pomeranz, Miriam Keltz. / Disseminated superficial actinic porokeratosis. In: Dermatology Online Journal. 2012 ; Vol. 18, No. 12. pp. 24.
@article{6dab048622c44b119b5de86cf03a10fb,
title = "Disseminated superficial actinic porokeratosis",
abstract = "Disseminated superficial actinic porokeratosis, which was described in 1966, is characterized by small, atrophic patches with distinctive keratin rims that occur on sun-exposed areas of the extremities, shoulders, and back. The diagnosis is based on the histopathologic finding of a cornoid lamella, absence of a granular layer, and often a thin epidermis. It is associated with exposure to ultraviolet radiation. Gene studies suggest a pathway defect in which several mutations in keratinocyte proliferation and differentiation lead to development of porokeratosis.",
author = "Panta Rouhani and Max Fischer and Shane Meehan and Pomeranz, {Miriam Keltz}",
year = "2012",
month = "12",
language = "English (US)",
volume = "18",
pages = "24",
journal = "Dermatology Online Journal",
issn = "1087-2108",
publisher = "Department of Dermatology UC Davis",
number = "12",

}

TY - JOUR

T1 - Disseminated superficial actinic porokeratosis

AU - Rouhani, Panta

AU - Fischer, Max

AU - Meehan, Shane

AU - Pomeranz, Miriam Keltz

PY - 2012/12

Y1 - 2012/12

N2 - Disseminated superficial actinic porokeratosis, which was described in 1966, is characterized by small, atrophic patches with distinctive keratin rims that occur on sun-exposed areas of the extremities, shoulders, and back. The diagnosis is based on the histopathologic finding of a cornoid lamella, absence of a granular layer, and often a thin epidermis. It is associated with exposure to ultraviolet radiation. Gene studies suggest a pathway defect in which several mutations in keratinocyte proliferation and differentiation lead to development of porokeratosis.

AB - Disseminated superficial actinic porokeratosis, which was described in 1966, is characterized by small, atrophic patches with distinctive keratin rims that occur on sun-exposed areas of the extremities, shoulders, and back. The diagnosis is based on the histopathologic finding of a cornoid lamella, absence of a granular layer, and often a thin epidermis. It is associated with exposure to ultraviolet radiation. Gene studies suggest a pathway defect in which several mutations in keratinocyte proliferation and differentiation lead to development of porokeratosis.

UR - http://www.scopus.com/inward/record.url?scp=84871497002&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=84871497002&partnerID=8YFLogxK

M3 - Article

VL - 18

SP - 24

JO - Dermatology Online Journal

JF - Dermatology Online Journal

SN - 1087-2108

IS - 12

ER -