Direct, interferon-independent activation of the CXCL10 promoter by NF-κB and interferon regulatory factor 3 during hepatitis C virus infection

Agriculture & Biology

• interferon regulatory factor-3 100%
• Hepatitis C virus 78%
• interferons 61%
• promoter regions 38%
• Toll-like receptor 3 31%
• infection 28%
• hepatocytes 21%
• retinoic acid 21%
• binding sites 15%
• response elements 14%
• chemokine CXCL10 11%
• Toll-like receptor 5 10%
• polyinosinic-polycytidylic acid 8%
• genes 8%
• luciferase 6%
• species recruitment 5%
• binding proteins 5%
• transcription factors 4%
• transcription (genetics) 4%
• RNA 3%
• genotype 3%
• mutants 3%

Medicine & Life Sciences

• Interferon Regulatory Factor-3 79%
• Hepacivirus 50%
• Interferons 49%
• Virus Diseases 39%
• Toll-Like Receptor 3 25%
• Hepatocytes 20%
• Tretinoin 18%
• Binding Sites 13%
• Response Elements 13%
• poly(U-C) 11%
• interferon type III 9%
• Genes 9%
• Toll-Like Receptor 5 9%
• Chemokine CXCL10 8%
• CCAAT-Enhancer-Binding Proteins 8%
• Protein C 6%
• Transcription Factor AP-1 6%
• Luciferases 5%
• Transcription Factors 4%
• Genotype 4%
• RNA 4%
• In Vitro Techniques 2%