TY - JOUR
T1 - Color-Naming Defects in Dyslexic Boys
AU - Denckla, Martha Bridge
N1 - Funding Information:
I This work was supported in part by Research Grant R-6822 ("Neurological Factors in Dyslexia," Richard L. Masland, principal investigator) of the Easter Seal Research Foundation for Crippled Children and Adults.
PY - 1972
Y1 - 1972
N2 - Five boys, aged 7.5–10.7 years, presented with a syndrome of severe dyslexia and color-naming difficulty, without comparable object or picturenaming difficulty, without color blindness, and without conspicuous “soft” neurological signs. Their color-naming defect was compared to a variety of color anomias reported in adults with acquired lesions and it was concluded that these boys most closely resemble the “aphasic”, rather than the “disconnexion” type. The remarkable severity and purity of their dyslexia, coupled with the color-naming defect, suggests that a highly specific differentiation of function exists within the cerebral language areas and discrete malfunction can account for specific subtypes of dyslexia.
AB - Five boys, aged 7.5–10.7 years, presented with a syndrome of severe dyslexia and color-naming difficulty, without comparable object or picturenaming difficulty, without color blindness, and without conspicuous “soft” neurological signs. Their color-naming defect was compared to a variety of color anomias reported in adults with acquired lesions and it was concluded that these boys most closely resemble the “aphasic”, rather than the “disconnexion” type. The remarkable severity and purity of their dyslexia, coupled with the color-naming defect, suggests that a highly specific differentiation of function exists within the cerebral language areas and discrete malfunction can account for specific subtypes of dyslexia.
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U2 - 10.1016/S0010-9452(72)80016-9
DO - 10.1016/S0010-9452(72)80016-9
M3 - Article
C2 - 5043791
AN - SCOPUS:0015357348
SN - 0010-9452
VL - 8
SP - 164
EP - 176
JO - Cortex
JF - Cortex
IS - 2
ER -