Cardiopulmonary effects of high thoracic epidural anesthesia

Research output: Contribution to journalArticle

Abstract

Thoracic epidural anesthesia (TEA) is an excellent method for providing segmental anesthesia and analgesia in the thoracic and upper abdominal regions. By causing extensive though incomplete sympathetic denervation, TEA significantly depresses cardiac performance by affecting sinus nodal and atrioventricular conduction function as well as myocardial contractility. TEA also reduces myocardial oxygen consumption, dilates stenotic segments of coronary arteries, reduces infarct size, and speeds up the recovery of stunned myocardium and perhaps provides myocardial protection, which makes it quite useful in the management of patients with severe symptomatic ischemic cardiomyopathy. By providing selective analgesia, TEA improves inspiratory flows and capacities thus yielding a beneficial effect to counteract postoperative atelectasis. Its effects on peak expiratory flows and pressures may, however, affect the effectiveness of coughing. TEA does not correct or improve postoperative diaphragmatic dysfunction. Finally, concerns that thoracic sympathectomy may induce bronchospasm in patients with hyperreactive airways or induce hypoxia by inhibiting hypoxic pulmonary vasoconstriction [HPV] are unfounded.

Original languageEnglish (US)
Pages (from-to)56-66
Number of pages11
JournalTechniques in Regional Anesthesia and Pain Management
Volume2
Issue number1
DOIs
StatePublished - Jan 1 1998
Externally publishedYes

ASJC Scopus subject areas

  • Anesthesiology and Pain Medicine

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