Calcific neurocysticercosis and epileptogenesis

Theodore E. Nash, O. H. Del Brutto, J. A. Butman, T. Corona, A. Delgado-Escueta, R. M. Duron, C. A.W. Evans, R. H. Gilman, A. E. Gonzalez, J. A. Loeb, M. T. Medina, S. Pietsch-Escueta, E. J. Pretell, O. M. Takayanagui, W. Theodore, V. C.W. Tsang, H. H. Garcia

Research output: Contribution to journalReview articlepeer-review

Abstract

Neurocysticercosis is responsible for increased rates of seizures and epilepsy in endemic regions. The most common form of the disease, chronic calcific neurocysticercosis, is the end result of the host's inflammatory response to the larval cysticercus of Taenia solium. There is increasing evidence indicating that calcific cysticercosis is not clinically inactive but a cause of seizures or focal symptoms in this population. Perilesional edema is at times also present around implicated calcified foci. A better understanding of the natural history, frequency, epidemiology, and pathophysiology of calcific cysticercosis and associated disease manifestations is needed to define its importance, treatment, and prevention.

Original languageEnglish (US)
Pages (from-to)1934-1938
Number of pages5
JournalNeurology
Volume62
Issue number11
DOIs
StatePublished - Jun 8 2004

ASJC Scopus subject areas

  • Clinical Neurology

Fingerprint Dive into the research topics of 'Calcific neurocysticercosis and epileptogenesis'. Together they form a unique fingerprint.

Cite this