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Biological significance and molecular mechanisms of p53-induced apoptosis
A. Bedi
School of Medicine
Research output
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Contribution to journal
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Article
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peer-review
13
Scopus citations
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Chemical Compounds
Oncogenic
100%
Genotoxic
84%
Protein Hydrolysis
77%
Cell Death
76%
Reactive Oxygen Metabolite
66%
Antineoplastic Agent
63%
Mutation
60%
Chemical Transformation
44%
Protein
29%
Medicine & Life Sciences
Apoptosis
43%
Neoplasms
23%
Death Domain Receptors
21%
Tumor Necrosis Factor Receptors
20%
Caspases
18%
Tumor Suppressor Genes
17%
Cell Cycle Checkpoints
17%
Antineoplastic Agents
17%
Oxidation-Reduction
17%
Proteolysis
16%
Reactive Oxygen Species
14%
Cell Cycle
14%
Hypoxia
14%
Cell Death
13%
Transcription Factors
13%
Mutation
9%
Genes
7%
Proteins
6%
Therapeutics
2%