Arsenic and old FLT3

Research output: Contribution to journalReview articlepeer-review


FLT3-internal tandem duplication (FLT3-ITD) mutations are common in acute promyelocytic leukemia (APL) and render the disease more difficult to manage or cure. In this issue of Blood, Esnault et al begin to unravel how the mutation-activated FLT3 receptor impedes the effects of all-trans retinoic acid (ATRA) and how arsenic counters this.

Original languageEnglish (US)
Pages (from-to)1392-1393
Number of pages2
Issue number13
StatePublished - Mar 2019

ASJC Scopus subject areas

  • Biochemistry
  • Immunology
  • Hematology
  • Cell Biology


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